Past awards

Edition 2021 (COVID-19)

Grant: 75 000 Euro

- Theme: Impact of COVID-19 on patients and/or ART services
- Recipient:

From individual to clinic to population: the economic, public health and regulatory consequences of the response to COVID-19 pandemic on IVF in Europe
Scott Nelson
University of Glasgow (UK)

Grant: 200 000 Euro

- Theme: Impact of SARS-CoV-2 on reproductive tissues, reproductive cells and/or (ART) pregnancy
- Recipient:

The effects of SARS-CoV-2 infection in human pre-implantation embryos
Manuel Viotti
Zouves Foundation for Reproductive Medicine (USA)


Edition 2020

Grant: 75 000 euro

- Theme: Sperm Freezing and Function
- Recipient:

Fertility preservation in (peri)pubertal boys: Developing an approach for simultaneous cryopreservation of sperm and spermatogonial stem cells from testicular biopsies.
Rod Mitchell
University of Edinburgh (UK)

Grant: 200 000 euro

-Theme: Protecting and Treating Male Fertility

Exploiting multi-omics to assess and map the fertility potential of cryopreserved prepubertal testicular tissues
Anne Goriely
University of Oxford (UK)


Edition 2018

Grant: 50 000 euro

- Theme: Chromosome and DNA integrity of oocytes and embryos
- Recipient:

Probing actin-dependent chromosome cohesion in mammalian oocytes
Binyam Mogessie
University of Bristol (UK)

Grant: 150 000 euro

- Theme: Maternal determinants of embryo developmental ability
- Recipient:

Embryo quality revisited: understanding and treating the hidden impact of obesity
Roger Sturmey
University of Hull (UK)


Edition 2016

Grant: 50 000 euro (project finalised)

- Theme: endometrial receptivity
- Recipient:

Uterine fluid exosomes as a “liquid biopsy” for depicting personalized endometrial receptivity
Paola Vigano
Ospedale San Raffaele, Milano (Italy)

- Summary:
On the largest sample size of human Extracellular Vesicles (EVs) ever analyzed with RNA-sequencing, this study establishes a gene signature to use for less-invasive endometrial receptivity tests. Our results are indeed the first to show that the transcriptome of UF-EVs correlates with the endometrial tissue transcriptome, that RNA signatures in uterine fluid (UF)-EVs change with endometrial status, and that UF-EVs could serve as a reservoir for potential less-invasive collection of receptivity markers. These thus represent a step forward in the design of less-invasive approaches for real-time monitoring of endometrial status, necessary for advancing the field of reproductive medicine.

- Relevant publication(s):

Grant: 150 000 euro (project finalised)

- Theme: endometrial receptivity
- Recipient:

Elucidating a new mechanism of endometrial receptivity establishment in women and its clinical significance
Guiying Nie
Monash University (Australia) and Vrije Universiteit Brussel (Belgium)

- Summary:
Podocalyxin is a key and previously unknow negative regulator of human endometrial epithelial receptivity. It is expressed at the apical surface of all epithelial and endothelial cells within the human endometrium but is specifically down-regualted in the luminal epithelium at receptivity. This down-regulation is likely mediated by progesterone and would selectively convert the endometrial surface from a non-receptive to an implantation-permitting state. Epithelial podocalyxin may provide a new parameter for optimizing and evaluating endometrial receptivity.

- Relevant publication(s): and

Edition 2014

Grant: 150 000 euro (project finalised)

- Theme: none
- Recipient:

Can tyrosine kinase inhibitors protect the ovary against chemotherapy-induded damage?
Norah Spears
Universities of Edinburgh (UK) and Rome Tor Vergata (Italy)

- Summary:
Our work has shown that a novel ovarian culture method can be used to carry out large-scale screening to test drug effects on the ovary, with the method used to systematically testing a bank of tyrosine kinase inhibitors. Results show that GSK-3 inhibitors are best able to protect the ovary against cisplatin-induced damage, demonstrating that they protect follicle health, with GSK3-β involved in cisplatin-induced oocyte apoptosis.

- Relevant publication(s):